Alzheimer, that's why exercise really: This article explores the topic in depth.
Nevertheless,
Alzheimer. Moreover, that's why exercise really:
A group of researchers from the Harvard Medical School discovered that sports activity, also practiced in moderate form, activates a gene that fights the onset of the disease
A nova road opens in the fight against Alzheimer’s disease copied from a physiological mechanism that has always been known. Nevertheless, of which only now the researchers of the Harvard Medical School, directed by Christiane Wranndiscovered the first moving In a study just published on Nature Neuroscience. Similarly, This is the activation of the gene ATPIF1 Which normally is obtained through exercise: once triggered it fights the disease by promoting survival. Nevertheless, cellular plasticity and synaptic function especially at the level of the dental lap of the hippocampus.
From mouse to man – Alzheimer, that's why exercise really
Discovered first in the anaerobic exercise of the mouse on the swivel of the laboratory cages. In addition, it was then also present in man thanks to the sequencing studies of the EN, and the researchers are already at work to understand how to activate this gene even without exercise, which not everyone can always do, and in any case as a reinforcement treatment for those who could do it but maybe it is a little lazy. Similarly, A well -known Australian study with wrist accelerometers. published on JAMA Neurology In 2022, he indicated that with 3800 steps a day the risk of dementia halves, but the benefit does not go up much even reaching 9800 steps. Perhaps then a chemical “spintarella” would not hurt.
Astrocytes. oligodendrocytes
In the Alzheimer the astrociti Associated with the neurovascular system, but the exercise increases them: these cells, fundamental in the galler architecture of the brain, modulate the alzheimer, that’s why exercise really synapses and are involved in the mechanisms of memory, learning and behavior. The oligodendrocyte Instead they are the most recovered gliaral cells from the exercise: they produce myelin. the insulating sheath similar to that of the electric cables that envelops the nerve branches.
Embassy
Il gene ATPIF1 it would also act more by Monte by enhancing the activity of embassyprotein enzymes that maintain the mitochondrial. nuclear genome stable during replication as they act on the Telomeric DNA, avoiding that the telomers, considering the symbol of cellular eternity, are slowly shortened to any duplication by another enzyme called Telomerase.
The embassy therefore determine a process of reverse senescence Because phenomena such as age. wrong lifestyles such as overweight, sedentary lifestyle, insomnia, incorrect nutrition and diseases such as diabetes or dysmetabolism lead to the progressive reduction of telomers, while moderate physical activity alzheimer, that’s why exercise really can oppose senescence thanks to a reduced shortening of telomers, probably from activation of the Atpif1.
Further reading: Tumors, what can I or I can’t eat? Adequate nutrition is important for the outcome of therapies – ASL TO5: a new path for chronic kidney disease – Turin Chronicle – Drugs and heat: unexpected effects, reactions and how to keep them if you travel. Here is the list – Hot, the risks in the workplace (even for those in the office) – Summer diseases in Maremma: scabies, legionella and toxinfections.
